Taken together , a possible mechanism for sa improving al - tolerance of c . tora is proposed : treatments with sa promoted the al - induced increase in citrate efflux , and this resulted in the decreased accumulation of al in root tips , thereby causing a limited oxidative stress and root growth inhibition Sa缓解c . tora铝毒害的可能机制：增加柠檬酸的分泌，降低根尖细胞内al的含量，减轻铝诱导的氧化损伤，从而增强c . tora对铝毒的抗性。
Light microtechnique and sa - galactosidase method was used to study the effects of super - high - concentration of glucose on the senescence of human diploid fibroblast 2bs cells , ros and the membrane potential of mitochondria during this process were measured . our results showed that 200 mmol l of glucose inhibited the growth of 2bs cells , led to the changes of reactive oxygen species and decrease of mitochondrial membrane potential , and caused senescence of 2bs cells rapidly . it supports the hypothesis of oxidative damage of senescence . moreover it is a better system for the study of the effects of ros during the process of replicative senescence 利用光学显微镜观察和酸性-半乳糖苷酶染色技术研究了高浓度葡萄糖对人二倍体成纤维细胞2bs细胞衰老进程的影响，并用流式细胞仪检测了此过程中活性氧和线粒体膜电位差的变化。结果表明： 200 mmol l的葡萄糖对2bs细胞有生长抑制作用，能引起活性氧含量的变化，导致线粒体膜电位差显著下降，并诱导了细胞的衰老。这为氧化损伤假说提供了新的证据，并为研究活性氧和复制衰老之间的关系提供了较好的体系。
The expression of tstrx from different tissue also shows different . these results show that this gene is a stress response gene or plays an important role in slat stress tolerance . aiming to identify the functions of this gene and do some further study , we have cloned the gene into the agrobacterium tumefaciens binary vector prok ii and pcambia 3013 结合实验结果以及用200mmol . l ~ ( - 1 ) nacl处理的小盐芥地上部分构建的zap - cdna文库中得到的编码硫氧还蛋白的cdna序列的相关资料推测： tstrx基因可能在保护盐芥免受因胁迫引起的氧化损伤中起作用。
In conclusion , the method of enzymolysis combined with hplc to extract cp i from pigskin was proposed . this method showed the adventages of high performance , high selectivity and easily amplification . acoording to the experiments of identifying the physico - chemical property and the biosafety of cp i , it was concluded that cp i had high bioactivity and no toxicity effect , and that cp i had the ability to protect cells from oxidative damnification 综上所述，利用酶解和高效液相色谱制备相结合的方法可以从猪皮中分离纯理学硕士学位论文: i型胶原蛋白的制备、鉴定及其抗uva氧化损伤作用的初步研究化cpi ，该方法具有高效性，高选择性和易放大性;理化性质鉴定及生物安全性试验表明，该物质为纯活性胶原蛋白，无毒副作用;同时对体外细胞的uva氧化损伤具有较好的保护作用。
Glutathione redox system as the important parts of defense systems was studied after exposure to sulfur dioxide at different concentrations . dna damage caused by so2 and lead acetate were also studied . protective roles of seabuckthorn seed oil on oxidative damage caused by so2 in liver and lung of mice were also investigated 本文在整体动物水平上研究了不同浓度二氧化硫吸入后小鼠组织谷胱甘肽氧化还原系统( glutathioneredoxsystem )的动态变化规律；二氧化硫所致氧化损伤的防护作用研究；二氧化硫和大气颗粒物中亲气性重金属元素铅对dna的联合损伤作用。
In this paper , how to prepare cp i and identify physico - chemical property had been studied , and uva oxidative damage l929 was perliminary discussed . these datas provided theoretic foundation and scientific proof for further research and development of this product . pigskin was used as material 本文围绕型胶原蛋白( cp )的制备及其理化性质进行研究，同时在其防止长波紫外线( uva )对细胞的氧化损伤方面进行了初步探索，为cp的进一步研发提供理论基础和科学依据。
In preventive groups , the levels of lpo in liver and lung were decreased and the activities of gst were increased compared with those in so2 inhalation group . with higher dosage of seabuckthorn seed oil administrated introperitoneally ( 6ml / kg body weight , 8ml / kg body weight ) , lpo and gst changed significantly . it was showed that seabuckthorn seed oil treatment born more protective roles to lung than liver 通过腹腔注射沙棘油对小鼠抗二氧化硫所致氧化损伤作用的研究表明：注射沙棘油后， so _ 2吸入组小鼠的肝、肺组织脂质过氧化物水平( lipidperoxidation ， lpo )都有不同程度的降低， gst活性均有不同程度的升高，且沙棘油高剂量组(分别相当于6ml沙棘油kg体重， 8ml沙棘油kg体重)的变化要大；肺组织的变化比肝脏组织显著，即沙棘油对肺脏的保护作用较肝脏明显。
