Conclusion pqs can protect myocardium from ischemic injury in rats after ami by way of promoting angiogenesis in the infarcted or ischemic area of myocardium and upregulating expressions of vegf and bfgf in myocardial cells 结论pqs可增强急性心肌梗死大鼠心肌细胞vegf bfgf的表达，促进梗死及缺血区血管新生，发挥保护心肌的缺血性损伤作用。
Then the gray value of bfgf increased slowly and reached the peak point in the group of 10 . 5 - 11 . 5 d . 3 . in the group of 10 - 14 h , the gray value of cox2 positive nerve cells began to decrease obviously ( p < 0 10 1均组coxz阳性神经细胞的灰度值开始显著不久p一n ，至排一月h组coxz灰度值达最乡与其各实验主卜以一入了显名差异（ o
By coating the collagen solution containing bone morphogenic protein ( bmp ) or basic fibroblast growth factors ( bfgf ) on the plla membrane surface via the " grafting and coating " method , bioactive plla membranes were prepared 进一步利用“接枝涂层”技术，将含有生长因子bmp或bfgf的胶原溶液涂层在plla膜表面，制备了含有生长因子的活性plla材料。
But many facts show that some growth factors can also induce apoptosis , for example , epidermal growth factor ( egf ) can induce apoptosis of a431 , mda - mb - 468 cell line , bfgf have the same effect on mcf - 7 cell line and e wings ' sarcoma 但许多证据显示，某些生长因子也能够诱发细胞凋亡，如表皮生长因子( egf )能够诱导a431 、 mda - mb - 468等细胞发生凋亡。
With the previous relative research , we suppose that there was a common signal transduction pathway between shear stress and lps . the inhibit effect of shear stress do not bring into play via increasing the level of bfgf 我们通过已有的相关研究推断，剪切力对lps诱导凋亡的抑制效应可能是由于剪切力与lps的信号传导中存在共同的通路，而不是由于剪切力诱导bfgf分泌抑制凋亡的。
Over the induction process cell body became increasing spherical and retractile , exhibiting a typical neuronal perikaryal appearance . western blot a - nalysis indicated that cells exhibited increased expression of the neuronal marker nse after 5h of bfgf , atra treatment 免疫细胞化学显示在加人bme5小时后部分细胞胞体收缩变圆，胞质呈nse染色强阳性，细胞呈有较长突起的双极，多极型。
