vasospasm造句

• Delayed cerebral vasospasm after subarachnoid hemorrhage
  蛛网膜下腔出血后迟发性脑血管痉挛
• Risk factors for cerebral vasospasm following subarachnoid hemorrhage
  蛛网膜下腔出血后脑血管痉挛的危险因素
• Investigation of apoptosis in the mechanism of cerebral vasospasm after subarachnoid hemorrhage
  细胞凋亡对蛛网膜下腔出血后脑血管痉挛发病机制的研究
• Current state and clinical progress on therapy of sequent cerebral vasospasm in subarachnoid hemorrhage
  蛛网膜下腔出血后脑血管痉挛的治疗现状及临床进展
• Different administrations of nimodipine for the treatment of cerebral vasospasm after subarachnoid hemorrhage
  尼莫地平不同给药途径治疗蛛网膜下腔出血后脑血管痉挛
• The subarachnoid hemorrhage from a ruptured aneurysm is more of an irritant producing vasospasm than a mass lesion
  因动脉瘤破裂造成的蛛网膜下腔出血主要引发血管痉挛,其次才是团块损害。
• Coronary artery vasospasm can cause a transient , abrupt , marked decrease in the diameter of an epicardial coronary artery
  摘要冠状动脉血管痉挛能造成一个暂时的，突然的，显着的心外冠状动脉内径的减少。
• Allen jm , et al . presence of neuropeptide y in humem circle of willis and its possible role in cerebral vasospasm . lancet 1984 , 2 550
  季鹰，吴建中，赵雅度.神经肽y在蛛网膜下腔出血引起脑血管痉挛中的作用.中国病理生理杂志， 1991 ， 7 ( 4 ) 412 415
• The mechanism of the cerebral vasospasm may be related to endothelin , endothelin - derived constricting factor , endothelin - derived relaxating factor and nitric oxide
  其发生机制可能与内皮素、内皮衍生收缩因子、内皮衍生舒张因子和一氧化氮等因子有关。
• The author also discussed the methods of the animal model of the subarachnoid hemorrhage and the effect of endothelin receptor on the cerebral vasospasm caused by subarachnoid hemorrhage
  本文讨论了实验性蛛网膜下腔出血动物模型的制作方法，内皮素受体在蛛网膜下腔出血诱发脑血管痉挛中的作用。
• Conclusion : cerebral infarction was strongly associated with poor outcome after aneurysmal sah . the most important potentially treatable factor associated with infarction was symptomatic vasospasm
  结论：脑梗与动脉瘤性sah后的不良预后显著相关。症状性血管痉挛是最重要的与脑梗相关的有可能被治愈的因素。
• The cerebral vasospasm is the main complication caused by subarachnoid hemorrhage , and may induce the elevation of the intracranial pressure , decrease of the cerebral blood flow and even death
  摘要蛛网膜下腔出血的主要并发症是脑血管痉挛，脑血管痉挛可引起颅内压升高、脑血流降低和脑灌注压降低，甚至死亡。
• Clinically significant vasospasm requiring angioplasty occurred in 11 patients ( 6 . 5 % ) with subarachnoid hemorrhage , and 120 patients ( 71 % ) with subarachnoid hemorrhage required ventricular shunts
  临床上出现明显的血管痉挛并伴有蛛网膜下腔出血，需血管成形术治疗者11名（ 6 . 5 % ） ， 120名（ 71 % ）有蛛网膜下腔出血者行脑室分流术。
• Multivariable analysis found that cerebral infarction was significantly associated with increasing patient age , worse neurological grade on admission , history of hypertension or diabetes mellitus , larger aneurysm , use of prophylactically or therapeutically induced hypertension , temperature more than 38 degrees c 8 days after sah , and symptomatic vasospasm
  多因素分析发现脑梗的发生与病人年龄增加、入院时较差的神经学分级、有高血压或糖尿病史、较大的动脉瘤、预防性或治疗性高血压的应用、 sah后8天体温高于38度和症状性血管痉挛显著相关。
• Cerebral vasospasm is a common and devastating medical complication of subarachnoid hemorrhage , which has high morbidity and mortality . however ; the mechanisms of cerebral vasospasm after subarachnoid hemorrhage still remain unknown , which is mainly related to five aspects including vasomotion factor , hemolysate , structure of vessel wall , inflammatory reaction and abnormal gene expression . the article describes its research development basing on this five aspects
  脑血管痉挛是蛛网膜下腔出血常见而又严重的并发症,有很高的致残率和致死率,但是脑血管痉挛的发生机制目前仍不清楚,其内容主要涉及血管舒缩因子、溶血产物、血管壁结构、炎性反应、基因异常表达5个方面,本文从这五个方面分别介绍了其研究进展。